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Roxane Holt, a maternal-fetal medication fellow and co-lead author of the scholarly study. ‘When patients present in preterm labor, we don’t have a whole lot of therapy to stop the labor,’ she said. UT Southwestern experts compared preterm birth versions in mice. They injected lipopolysaccharide to market infection-like circumstances and an inflammatory response in a single model. In the other, they administered mifepristone to simulate the withdrawal of the gestation-supporting hormone progesterone, which usually takes place at the end of a pregnancy. Researchers statement that cervical adjustments in inflammation-induced circumstances are caused by an influx of white bloodstream cells and an increased expression of pro-inflammatory markers without increase in the expression of genes induced in term ripening.